Oct

31

The patient was a 59-year-old woman with SLE who had developed multiple nodules on the neck and face over several years. Because of major renal insufficiency, she also had been receiving hemodialysis 3 times per week (3.5 hours) for >10 years. The first clinical differential diagnoses were cutaneous SLE, nephrogenous dermatopathy, calciphylaxis, and calcinosis.
The clinical picture was obscured by secondary inflammations and ulcerations caused by self-inflicted trauma.
Multiple sampling attempts by cutaneous core biopsies resulted in histologic diagnosis of unspecific, secondary inflammatory changes. Deep surgical excision of 1 subcutaneous nodule on the scalp indicated subcutaneous helminthosis.
The patient was treated with ivermectin and subjected to 2 plastic surgeries for facial reconstruction, after which she recovered.

Oct

31

From Here :

There has been considerable speculation concerning possible differences in the ecology of the genospecies and most data suggest that B. afzelii is associated with rodents, B. garinii mainly with birds and B. valaisiana exclusively with birds.
B. garinii has also been identified in tissues taken from rodents and at least one strain of B. garinii (serotype 4), rarely found in ticks but associated with neuroborreliosis, appears to be specific for rodents.
In one study of double infections in ticks, B. valaisiana was most often associated with B. garinii, and in another study both these species were shown to be transmitted from blackbirds (Turdus merula) to ticks. B. burgdorferi s.s. seems to occur frequently in both birds and rodents.
Most recently B. lusitaniae has been strongly associated with lizards in two separate studies.

Oct

26


Numbers of these parasites are at an all time high, with environmental health officers reporting a trebling in the number of cases over the past five years, putting humans and their pets at risk of infection.Ticks can carry Lyme disease.

Vets have reported a significant upsurge in the number of cases and claim dirty homes, busy lifestyles and general ignorance are also contributory factors.

Experts are warning that the problem is set to worsen over the next few weeks as this autumn is likely to provide the ideal conditions for the disease carrying parasites, creating serious implications for human health. Ticks can carry Lyme disease, a paralysing blood disease which can cause blindness and even death.

A response to this article :

I have lived in the country for all of my 60 years and worked with animals throughout. I have seen a badger (dead) with over two hundred ticks and have literally removed the same number from my dogs and until this year not really taken them seriously. At the beginning of August I was the host to a deer tick, which overstayed it’s welcome. At the first opportunity, (6 days) I visited the local GP who correctly prescribed anti biotics, having looked at his computer for some time, admitting at the end of the consultation that “This is a new one for me. I experienced the usual lethargy, headaches (permanent)aching limbs and stiffnes of some joints. The anti biotics seemed to stabilise the symptoms but they did not go away. Four weeks to the day I returned to the GP, not the same one, and explained my symptoms again. After a long very silent pause, I asked for blood tests to be taken, which was agreed. Three weeks later I returned, by my own volition to the second GP, who explained the tests had returned negative and that “The symptoms are in my head, take paracetamol for the headache.” I immediately asked for a referral to the Clinic for Tropical Diseases in Liverpool, which the stunned GP agreed to. The point of all of that is not to be put down by the GP as very little is known of the problems that arise from a tick bite and in my case very little interest was shown by the second GP.

Some ten weeks later I find that my extremely active life has nown changed beyond all recognition. I have a permanent headache which I now tolerate, extreme fatigue which hits without warning, stiffening of the joints, muscle spasm (possibly caused by a reduction in my activity level) dry chesty cough and I now speak with a very annoying nasal twang.

I have had numerous blood tests, taken by the GP and the clinic in Liverpool, these are to be repeated monthly in order to identify whether I have Lyme’s Disease or Tick Borne Encephilitis which have similar symptoms. I am told there is no cure for either, but Lyme’s can be controlled with rotational anti biotics.

Your article was interesting, in that it highlighted the number of possible cases that are not reported. How many patients have been sent packing with the notion that the symptoms are simply imagined.

I have written to Aberdeen University where a study is being carried out by Dr.Bowman, that was two weeks ago and I await the courtesy of a reply.

I would be particularly interested to hear of a help group, or anyone who has, or thinks they have been bitten by either a sheep or deer tick and /or are experiencing the problems associated with this very unwelcome guest.

The moral here is pursue the GP relentlessly, as this is a very unpleasant experience and it can be with you for a very long time.

Oct

22

A fantastic page of links.

Oct

20

Garlic

October 20, 2007 | Leave a Comment

Garlic… From Here :

The crucial information from this article is, I think, “… That to maximize the health benefits of garlic, you should crush the bulbs at room temperature, and allow it to sit for about 15 minutes. This behaviour triggers an enzyme reaction that boosts the healthy compounds found in garlic.”

Oct

15

New Scientist

Oct

12

Although they were first considered as protozoa and later as viruses, chlamydiae are gram-negative obligate intracellular eubacteria. Originally, they were taxonomically categorised into their own order Chlamydiales, with one family, Chlamydiaceae, and a single genus, Chlamydia (Moulder et al. 1984). The genus included four species: C. trachomatis, C. psittaci (Moulder et al. 1984), C. pneumoniae (Grayston et al. 1989) and C. pecorum (Fukushi & Hirai 1992).

In 1999, it was recommended by Everett et al. (1999) that the genus Chlamydia should be divided in two genera, Chlamydia and Chlamydophila, containing altogether nine species (Table 3). In addition to the five new species, three new families (Parachlamydiaceae, Simniaceae and Waddliaceae) were also recommended. However, the proposal to change the taxonomic nomenclature for the Chlamyadiaceae family has not been generally accepted in the field (Schachter et al. 2001).

Two of the species, C. trachomatis and C. pneumoniae, are common pathogens in humans, whereas the other species occur mainly in animals. C. trachomatis has been isolated only from humans and comprises two human biovars (trachoma and lymphogranuloma venereum, LGV), including a total of 18 serovars, whereas C. pneumoniae has one human biovar (TWAR) and two animal biovars, one infecting horses (biovar equine) and the other infecting frogs and koalas (biovar koala).

From Here :

Oct

12

An in vitro study of the susceptibility of mobile and cystic forms of Borrelia burgdorferi to hydroxychloroquine.Brorson O, Brorson SH.
Department of Microbiology, Vestfold Sentralsykehus, Tonsberg, Norway.

In this work the susceptibility of mobile and cystic forms of Borrelia burgdorferi to hydroxychloroquine (HCQ) was studied.
The minimal bactericidal concentration (MBC) of HCQ against the mobile spirochetes was > 32 microg/ml at 37 degrees C, and > 128 microg/ml at 30 degrees C. Incubation with HCQ significantly reduced the conversion of mobile spirochetes to cystic forms.
When incubated at 37 degrees C, the MBC for young biologically active cysts (1-day old) was > 8 microg/ml, but it was > 32 microg/ml for old cysts (1-week old).
Acridine orange staining, dark-field microscopy and transmission electron microscopy revealed that the contents of the cysts were partly degraded when the concentration of HCQ was > or = MBC. At high concentrations of HCQ (256 microg/ml) about 95% of the cysts were ruptured. When the concentration of HCQ was > or = MBC, core structures did not develop inside the cysts, and the amount of RNA in these cysts decreased significantly.
Spirochetal structures inside the cysts dissolved in the presence of high concentrations of HCQ. When the concentration of HCQ was > or = MBC, the core structures inside the cysts were eliminated.
These observations may be valuable in the treatment of resistant infections caused by B. burgdorferi, and suggest that a combination of HCQ and a macrolide antibiotic could eradicate both cystic and mobile forms of B. burgdorferi.

PMID: 12102233 [PubMed - indexed for MEDLINE]

Plaquenil Info

Oct

12

It seems increasingly likely that the majority of Morgellons patients will test positive for Chlamidia Pneumoniae.
I have.
Indeed, as this is an airbourne pathogen, the majority of the population of this planet will have been exposed too.
It is interesting to note that air conditioning systems and sick buildings are frequently mentioned in lists of suspected catalysts of this illness.
Also, since the airline companies have effectively sealed the compartments of airplanes and recycled the air, any infections, passengers might be carrying, must be circulated to all of the people in the cabin too…..
Statistics indicate that over 50% of the world population has been exposed, but this figure is as high as 90% in Lyme Borreliosis patients.
Chlamidia Pneumoniae is also present in patients with Fibromyalgia, CF, CFS and host of other diseases.
I urge EVERYBODY to take some time to read the information to be found here.
It is essential reading.
This is an epidemic.

Oct

11

whitespots on arm

………..I would describe the skin symptom as white spots in the skin. Doctors like to call it mottled skin. It is some times as hard to see as this disease is to beat but if you pay close attention to your hands, forarms, and back side of your arms where you can see it best. I find it is easier to see in others from a distance than it is to see in yourself. I believe this is chlamydia……..

From Here:

Oct

11

….Meanwhile, in a private laboratory in Maryland, researchers were able to extract DNA from two fibre samples using protein-digesting enzymes. The DNA was sequenced and it was determined to resemble a type of fungus.

Many fungi embed roots and grow filaments, and this is consistent with the appearance of fibres found below the skin of Morgellons sufferers. The laboratory is applying for funding for further research…..

From Here :

Oct

9

As it mentions in this article, it is essential to understand this phenomenon.

From Here :

Oct

8

The protocol is :

Penicillin V 1gm 4 times a day,

Azithromycin 250mg half a tablet Mon/Wed/Fri

Ivermectin 12mg Day 1, 7 and 13

For 8 weeks then re-assess

Oct

8

flourmosi.jpg

BBC News :
A genetically modified (GM) strain of malaria-resistant mosquito has been created that is better able to survive than disease-carrying insects.

Oct

8

A Scotsman is in hospital after being bitten by a mosquito.

Oct

7

J Parasitol. 1984 Dec ;70 (6):963-6 6527192
A microfilaria of exceptional size from the ixodid tick, Ixodes dammini, from Shelter Island, New York.
P C Beaver , W Burgdorfer
Thirty or more microfilariae 0.70-1.32 mm in length were recovered from the hemocele of an unengorged adult tick, Ixodes dammini, that was collected from vegetation on Shelter Island, New York. Among approximately 500 I. dammini collected from the same area only 1 other was similarly infected. Outstanding features, in addition to size, were absence of a cephalic space and the presence of nuclei in 2 or 3 irregular rows extending to the end of a bluntly rounded tail. The microfilariae apparently were ingested in a blood meal that was taken when the ticks were larvae or nymphs, and had persisted alive without development.

Oct

6

William Harvey, MD, MS, MPH
Board Chairman, MRF

Sent: Wednesday, October 3, 2007 9:30:54 AM
Subject: Re: Treatment for Morgellons Disease

We are moving closer to understanding the mechanism of this illness and have almost completed the defining paper for submission to a medical journal for review. Early in 2007, we did a detailed study of 25 consecutive presumed Morgellons patients here, collecting over 407 parameters on each, from laboratory values, to physical exam findings, to detailed medical history. All parameters were mathematically collated to give us a first “look” at a Morgellons “average”. As the illness began with Ms Leitao’s search for a name for her son’s illness based solely on filaments she saw, all registrants have been purely self-diagnosed…again mainly based on the appearance of skin filaments, then later movement sensation.
The summary data was extremely revealing, and briefly, showed us consistent abnormalities in immune function, chronic systemic inflammation, multi-system involvement…and perhaps
what is turning out to be most important, the presence in all of actual parasites. By August 2007, we had micrographs of at least two genera and several species. The latter are so similar that we needed help in answering the question: exactly what species are they, as this impacts treatment significantly. As I speak, we have assembled a group of scientists at a state university laboratory with equipment capable of giving us the answer. In the group are three veterinarians, a parasitologist, a mycologist, an FBI trained forensics Nurse Practitioner, a PhD invertebrate zoologist, a psychiatrist experienced in infectious causes of behavioral change…and me. Hopefully, they will close the loop for us in the next few months.
Meanwhile, we knew enough months ago to resolve the illness in most, with two caveats. (1) Close to 90% get 90% well if we use intravenous antibiotics strictly tailored for one bacterium for several months. Hundreds have done this safely. The same antibiotics given orally take about 8 months. (2) With the finding of the most common parasite species, the use of one of two anti-helmenthic antibiotics will resolve many symptoms, especially the skin manifestation in days…but only in about half. Knowing the species in each individual may resolve this.All that said, treatment has to be guided carefully with regular testing of liver, kidney and marrow effects. A clinician must have extensive experience to avoid the errors we made. As we are
licensed physicians, it is not permissible for us to write treatment protocols on line. This isn’t done for control, but for patient safety and steering to a good outcome.
FYI, this phenomenon in NOT chronic Lyme disease, it’s not Chronic Fatigue Syndrome, and we are now certain it is NOT Delusions of Parasitosis. The latter in fact, will be the thrust of the
paper: the DOP label has resulted in inappropriate and incomplete treatment of countless people who never recovered. Once the final DNA sequencing is done and the paper completed, it will be published with treatment protocol(s) for all clinicians world wide to use. Meanwhile, as both primary organisms create brain limbic system abnormalities, we now
understand that the delusional component of the illness is real in many affected (but far from all), so correct psychotropic medication can help that component even if treated purely
independently. Nearly half the Morgellons are bipolar, but became so AFTER the parasite infection began. Others have formal diagnoses of extreme OCD or ADD/ADHD. The prevalence in children is no different from adults, and there appear to be no gender or race differences.
We know that effective treatment can be obtained in Colorado from physicians who worked with us in Colorado Springs.
Otherwise the globe is literally full of practitioners offering cures without factual basis. Our task is now focused: to complete and write the defining paper and provide the answer to everybody.

William Harvey, MD, MS, MPH
Board Chairman, MRF

October 2007
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