Feb

28

From Here :

All genetically eningeered crops contain bacterial DNA. This DNA contains a genetic element (the so called “CpG motif”) that stimulates the immune system to start a sequence of reactions leading to inflammation. Exposure to these genetic elements may lead to promotion of inflammation, arthritis and lymphoma (a malignant blood disease).

Furthermore, it has been demonstrated that DNA is not broken down in the gastro-intestinal tract to the extent formerly believed. Ingested DNA sequences large enough to contain whole genes have remained intact and entered the blood and tissues.

Feb

25

From Here :

The genus, Chlamydophilia, as obligate intracellular pathogens, induce chronic scarring in humans.
Chlamydia pneumoniae, a common cause of pneumonia, infects endothelial cells and circulating macrophages.
Evidence that C. pneumoniae is an opportunistic pathogen in chronic skin ulcers and other inflammatory skin conditions analogous to its role in atherosclerosis is reviewed……

The presence of viable C. pneumoniae in peripheral blood mononuclear cells suggests that C. pneumoniae may accompany these white blood cells to inflamed tissue sites and cause a secondary infection in the inflamed tissue.

Feb

25

From Here:

Diseases where an association has been discovered between chronic Chlamydia infection of body fluids and/or tissues with several disease syndromes of previously unknown etiology in humans which respond to unique antichlamydial regimens include:

Multiple Sclerosisi (MSi)
Rheumatoid Arthritis (RAi)
Inflammatory Bowel Disease (IBD)
Interstitial Cystitis (IC)
Fibromyalgia (FM)
Autonomic nervous dysfunction (AND neural-mediated hypotension);
Pyoderma Gangrenosum (PG)
Chronic Fatigue (CF) and Chronic Fatigue Syndrome(CFS).

Diseases where Cpn load has been associated where measured, and where treatment can create improvement in the primary condition:
Chronic hepatitis
Systemic lupus erythematosus
Arthritis
Thyroidosis
Scleroderma
Diabetes mellitus
Graves’ disease
Beschet’s disease and
Graft versus host disease (graft rejection).

Diseases where Cpn may be associated as a secondary or primary factor:

Sepsis syndrome
Cachexia
Circulatory collapse and shock resulting from acute or chronic bacterial infection
Acute and chronic parasitic and/or infectious diseases from bacterial
Viral or fungal sources such as a HIV, AIDS (including symptoms of cachexia, autoimmune disorders, AIDS dementiai complex and infectionsi) can be treated as well as Wegners Granulomatosis.

Various inflammatory diseases, there are certain features of the inflammatory process that are generally agreed to be characteristic. These include fenestration of the microvasculature, leakage of the elements of blood into the interstitial spaces, and migration of leukocytes into the inflamed tissue. On a macroscopic level, this is usually accompanied by the familiar clinical signs of erythema, edema, tenderness (hyperalgesia), and pain. Inflammatory diseases, such as chronic inflammatory pathologies and vascular inflammatory pathologies, including:
Chronic inflammatory pathologies such as aneurysms
Hemorrhoids
Sarcoidosis
Chronic inflammatory bowel disease
Ulcerative colitis
Crohn’s diseasei and vascular inflammatory pathologies
Disseminated intravascular coagulation
Atherosclerosis
Kawasaki’s pathology
Coronary artery disease
Hypertension
Stroke
Asthma
Chronic hepatitis
Multiple sclerosis
Peripheral neuropathy
Chronic or recurrent sore throat
Laryngitis
Tracheobronchitis
Chronic vascular headaches (including migraines
Cluster headaches and tension headaches) and pneumonia when demonstrated to be pathogenically related to Chlamydia infection.

Treatable disorders when associated with Chlamydia infection also include but are not limited to Neurodegenerative diseases including
Demyelinating diseasessuch as multiple sclerosis and acute transverse myelitis;
Extrapyramidal and cerebellar disorders such as lesions of the corticospinal system;
Disorders of the basal ganglia or cerebellar disorders;
Hyperkinetic movement disorders such as Huntington’s Chorea and senile chorea;
Drug-induced movement disorders such as those induced by drugs which block CNSi dopamine receptors;
Hypokinetic movement disorders
such as Parkinson’s disease;
Progressive supranucleo palsy;
Cerebellar and Spinocerebellar Disorders such as astructural lesions of the cerebellum;
Spinocerebellar degenerations (spinal ataxia)
Friedreich’s ataxia
Cerebellar cortical degenerations
Multiple systems degenerations (MencelDejerine-Thomas
Shi-Drager and Machado Joseph)); and systemic disorders (Refsum’s disease
Abetalipoprotemia, ataxia telangiectasia and mitochondrial multi-system disorder);
Demyelinating core disorders such as:
Multiple sclerosis
Acute transverse myelitis;
Disorders of the motor unit such as neurogenic muscular atrophies (anterior horn cell degeneration) such as
Amyotrophic lateral sclerosis
Infantile spinal muscular atrophy and juvenile spinal muscular atrophy);
Alzheimer’s diseasei;
Down’s Syndrome in middle age;
Diffuse Lewy body disease; Senile Dementia of Lewy body type;
Wernicke-Korsakoff syndrome;
Chronic alcoholism;
Creutzfeldt-Jakob disease;
Subacute sclerosing panencephalitis
Hallerrorden-Spatz disease; and
Dementia pugilistica

Malignant pathologies involving tumors or other malignancies such as:
Leukemias (acute chronic myelocytic
chronic lymphocytic and/or myelodyspastic syndrome);
Lymphomas (Hodgkin’s and non-Hodgkin’s lymphomas such as malignant lymphomas (Burkitt’s lymphoma or Mycosis fungoides));
Carcinomas (such as colon carcinoma) and metastases thereof;
Cancer-related angiogenesis;
Infantile hemangiomas;
Alcohol-induced hepatitis.
Ocular neovascularization
Psoriasis
Duodenal ulcers
Angiogenesis of the female reproductive tract
can also be treated when demonstrated by the diagnostic procedures described herein to be associated with Chlamydial infection.

An immunocompromised individual is generally defined as a person who exhibits an attenuated or reduced ability to mount a normal cellular or humoral defense to challenge by infectious agents
e.g., viruses, bacterial, fungi and protozoa. Persons considered immunocompromised include malnourished patients, patients undergoing surgery and bone narrow transplants, patients undergoing chemotherapy or radiotherapy, neutropenic patients, HIV-infected patients, trauma patients, burn patients, patients with chronic or resistant infections such as those resulting from myeloodysplastic syndrome, and the elderly, all of who may have weakened immunei systems. A protein malnourished individual is generally defined as a person who has a serum albumin level of less than about 3.2 grams per deciliter (g/dl) and/or unintentional weight loss greater than 10% of usual body weight.

The course of therapy, serological results and clinical improvements from compassionate antichlamydial therapy in patients diagnosed with the diseases indicated were observed and are reported in Example 5. The data provides evidence to establish that treatment of Chlamydia infection results in the serological and physical improvement of a disease state in the patient undergoing combination therapy. These observations were consistent among a variety of different diseases which fall within a generalized disease class.

Other Diseases of Unknown Etiology with New Evidence for a Chlamydia pneumoniae Etiology

Both C. trachomatis and C. psittaci exhibit a protean disease complex dependent on different serovars. One known basis for this diversity to date is the amino acid sequence of the MOMP. FIG. 1 shows a sequence alignment of various Chlamydia MOMPs. Note that the size and sequence are relatively homologous except for the four variable regions that are responsible for the serovar (serotype) basis of classification. Further, it has been discovered that C. pneumoniae infects blood vessel endothelial cells from which EBs are released in the blood stream. In addition, macrophages are known targets for C. pneumoniae and may serve as reservoirs and provide an additional mechanism of transmission. C. pneumoniae is thus able to spread throughout the human body, establishing infection in multiple sites and in multiple organ systems. Infected sites may exist for an extended period without inducing symptoms that are noticed by the patient or by an examining physician. Sequence variability of MOMPs or other chlamydial antigens may provide a basis for organ specificity while other chlamydial proteins, such as the 60K and 70K heat shock proteins or LPSi, may influence immune response.

C. psittaci and C. pecorum are known to cause a host of infections in economically significant animals. Thus, the teachings of this invention are relevant to animals. Throughout this application and for purposes of this invention, “patient” is intended to embrace both humans and animals. Virtually all rabbits and mice tested to date have PCRi signals for C. pneumoniae. They can be used as appropriate animal models for treatment using specific combination antibioticsi to improve therapy. (Banks et al., Ameri. J. of Obstetrics and Gynecology 138(7Pt2):952-956 (1980)); (Moazed et al., Am. J. Pathol. 148(2):667-676 (1996)); (Masson et al., Antimicrob. Agents Chemother. 39(9):1959-1964 (1995)); (Patton et al., Antimicrob. Agents Chemother. 37(1):8-13 (1993)); (Stephens et al., Infect. Immun. 35(2):680-684 (1982)); and (Fong et al., J. Clin. Microbiol. 35(1):48-52 (1997)).

Coupled with these developments are the recently developed rabbit models of coronary artery disease, where rabbits exposed to C. pneumoniae subsequently develop arterial plaques similar to humans (Fong et al., J. Clin. Microbiol. 35:48-52 (1997)). Most recently, a study at St. George’s Hospital in London found that roughly 3⁄4 of 213 heart attach victims have significant levels of antibodies to C. pneumoniae antibody and that those that have such antibodies achieve significantly lower rates of further adverse cardiac events when treated with antibiotics (Gupta et al., Circulation 95:404-407 (1997)). Taken together, these three pieces of evidence (the bacteria found in diseased tissue, inoculation with the bacteria causes diseases, and treating for the bacteria mitigates disease) make a case for a causal connection.

Feb

20

From Here :
With two major international airports and airplanes flying overhead day and night, it’s no surprise that London has one of the busiest flight paths anywhere in the world.

What might surprise you, though, is that those zigzag white lines across the sky, or contrails, as they’re known, might be having an adverse effect on London’s weather.

Inside Out investigates contrails and whether they could be bad for the capital’s climate….

How quaint …how, ermm, 20th Century…….ha ha ha ha ha…….. Contrails ?????….Reality Check Here…

Feb

17

Blue Nematode

February 17, 2008 | Leave a Comment

Or Fibre…..

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Feb

14

From Here :

I have no medical expertise and I claim none. I am not offering any medical advice or diagnosis with the presentation of this information. I am acting solely as an independent researcher providing the results of extended observation and analysis of unusual biological conditions that are evident.

There is increasing evidence that the general population may be affected by at least four pathogenic forms. Any perception that only a small segment of the population is affected by the so-called Morgellons condition may be quite false. Certain pathogenic forms are repeatedly showing up in a majority of human biological samples that have been observed, regardless of whether or not visible skin anomalies appear. The use of unusual skin conditions as a defining criteria of the existence of the “Morgellons condition” appears to be completely inadequate and it does not appear to encompass the severity, extent and distribution of the pathogenic forms.

The pathogenic forms are as follows:

1. An encasing or encapsulating filament, often barely visible to the human eye. This filament form often measures on the order of 12 to 20 microns in thickness. This bounding filament form usually contains within it a network of sub-micron fibers that are generally in parallel alignment with one another. This filament form has been found within airborne, skin, saliva and gum(dental) samples. It has also been observed within an anomalous hair sample. It appears possible that the function of this particular form is to deliver or encase a sub-network of additional pathogens of much smaller size. No natural identity of this form has been established.

2. A network of sub-micron filament forms. These are usually encased within the bounding filament referred to above. A human hair is on the order of 60-100 microns in diameter; an asbestos fiber is on the order of 2 microns in thickness. This pathogenic form has repeatedly been found within airborne, skin, saliva and gum(dental samples). This form has some morphological similarity to fungal forms (i.e., hyphae) but no suitable match to any known species exists at this time. In addition, there is no match at this time to a eukaryotic cell structure which is required for a match to fungus. A “budding”, or apparent growth structure composed of filaments of this same class has been identified on skin borne biological samples. A method for the testing of chitin, usually present in the cell walls of fungi, is to be established.1

3. A sub-micron spherical to oblate structure. The best size estimate for this form is currently on the order of 0.5 - 0.7 microns. These structures can and often do occur in large numbers within the biological samples. They have been found in isolation within the bounding filament as well as in large concentrations within the bounding filament. The have been found in combination with the sub-micron fibrous network within the bounding filament. They have been found in airborne, skin, saliva, gum(dental) and anomalous hair samples. They have been found within a broad distribution of human blood samples. They can and often do exist as an intracellular(within the cell) organism. The damage to cellular integrity(i.e, erythrocytes, or red blood cells) appears to correspond directly to the numbers present of the organism. The best assessment to date that can be offered by this researcher is that of a Chlamydia-like(coccus) bacterial form, with special a emphasis upon Chlamydia pneumoniae. The modification of conventional biological forms or of pathogens to exotic levels should be strongly considered throughout this investigation, in addition to the creation of new or unknown forms. The structure stains Gram-negative.

4. What is being called, for the time being, a “hybrid” form. This form has properties that are somewhat in between the sub-micron Chlamydia-like form and the sub-micron filament form. This form appears to be a state of transition between the two more defined sub-micron forms. It has been found only in biological samples and not in airborne samples. It occurs commonly within, but it is not restricted to, human blood samples and it often is in association with the Chlamydia-like form. It is of generally filamentous form but it is reduced in length compared to the sub-micron fibrous network that has been itemized. Mycoplasma or Mycoplasma variations or modifications are one consideration within this topic.

Feb

13

Cancer and Fungi

February 13, 2008 | Leave a Comment

From Here :

Genetics, the battle horse of modern oncology, is about to give up the ghost, together with its endless explanations based on enzymatic and receptor processes. Actually, it has already failed – it is just that no one can think of anything else that can take its place. The consequence of the oncological establishment’s inability to admit the failure of this line of research, which is at this point scientifically indefensible, is the continuous waste of a great quantity of economic, scientific and human resources.

What road to take? Where to look for those minimal logical elements that can shed light on the ignorance that pervades oncology?

Many thinkers – especially biologists – believe that by applying the Darwinian theory to the evolution of living beings, it may be possible to progress down a new path when it comes to the so-called degenerative diseases such as cancer, cardiopathies, and mental illness. According to this line of thought, these diseases are not attributable to environmental or genetic factors as is presently believed, but to infections.

Therefore, the answer to the question of what causes a degenerative disease can be found in the discipline that more than anything else has given luster to medicine, and which has promoted medicine from a mere practice to a science, that is microbiology.

It is in fact clear that, with the exception of bacteriology, the state of knowledge in this field of research is still quite limited, especially when it comes to viruses, sub-viruses and fungi, whose pathogenic valence, unfortunately, is little known.

It is true that scholars have given more attention to these biological entities recently, and in fact, the concept of “innocuous co-existence” attributed to many parasites of the body has begun to be questioned with much greater conviction. More determination is needed, however, in this process of the revision of microbiology so that the close connection between micro-organisms and degenerative diseases can be clarified.

I believe that it is by focusing on just one of these shadowy areas – on mycology, the realm of fungi – that it will become possible to discover the correct answers to questions concerning the problem of tumors.

Much evidence indicates that this is the road to take. The analogy between psoriasis – an incurable disease of the skin that many treat as fungus – and tumors, which are also an incurable disease of the organism, the symptomatological overlapping of systemic candidosis and cancer, and the strict genetic relationship between mycetes and neoplastic masses make this clear. These are all elements that support and confirm the point of view that all types of cancer, as happens in the vegetal world, are caused by a fungus.

A fungus infection – that of the Candida species – could supply the explanation for why a tumor occurs, and it is in this direction that research should move in the attempt to solve the problem of cancer once and for all.

Feb

10

maize.gif
From Here :

 

PARIS (AFP)–France officially imposed Saturday a ban on a strain of genetically modified corn produced by the U.S. agribusiness giant Monsanto Co. (MON), with the publication of an agriculture ministry order in the state’s official journal.

‘The growing of corn seeds … derived from genetically modified corn strain MON810 is prohibited on [French] territory’ read the order signed by Agriculture Minister Michel Barnier.

Feb

6

From Here :

………The other man attacked was the world’s leading lectins and plant genetic modification expert, UK-based Arpad Pusztai. He was vilified and fired from his research position at Scotland’s Rowett Research Institute for publishing industry-unfriendly data he was commissioned to produce on the safety of GMO foods.

His Rowett Research study was the first ever independent one conducted on them anywhere. He undertook it believing in their promise but became alarmed by his findings. The Clinton and Blair governments were determined to suppress them because Washington was spending billions promoting GMO crops and a future biotech revolution. It wasn’t about to let even the world’s foremost expert in the field derail the effort. His results were startling and consider the implications for humans eating genetically engineered foods.

Rats fed GMO potatoes had smaller livers, hearts, testicles and brains, damaged immune systems, and showed structural changes in their white blood cells making them more vulnerable to infection and disease compared to other rats fed non-GMO potatoes. It got worse. Thymus and spleen damage showed up; enlarged tissues, including the pancreas and intestines; and there were cases of liver atrophy as well as significant proliferation of stomach and intestines cells that could be a sign of greater future risk of cancer. Equally alarming - this all happened after 10 days of testing, and the changes persisted after 110 days that’s the human equivalent of 10 years.

GM foods today saturate our diet. Over 80% of all supermarket processed foods contain them. Others include grains like rice, corn and wheat; legumes like soybeans and soy products; vegetable oils; soft drinks; salad dressings; vegetables and fruits; dairy products including eggs; meat and other animal products; and even infant formula plus a vast array of hidden additives and ingredients in processed foods (like in tomato sauce, ice cream and peanut butter). They’re unrevealed to consumers because labeling is prohibited yet the more of them we eat, the greater the potential threat to our health.

Feb

3

From Here :

…..Also, it is important that you understand one of the founding principles of natural medicine…Herring’s Law of Cure. This law presents that your body will rid itself of anything unwanted (diseases, etc.) from top to bottom, from the inside to the outside, and in the reverse order in which it entered your system. As you will see, much of the work on my own body follows this law exactly.

February 2008
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