Mar

27

From Here :
Plastic waste in the oceans poses a potentially devastating long-term toxic threat to the food chain, according to marine scientists.
Studies suggest billions of microscopic plastic fragments drifting underwater are concentrating pollutants like DDT.
Most attention has focused on dangers that visible items of plastic waste pose to seabirds and other wildlife.
But researchers are warning that the risk of hidden contamination could be more serious.

“The thing that’s most worrisome about the plastic is its tenaciousness, its durability. It’s not going to go away in my lifetime or my children’s lifetimes.”

“The plastic washing up on the beach today… if people don’t take it away it’ll still be here when my grandchildren walk these beaches.”

Mar

20

From Here :

Epidemic confirmed
The researchers examined merged data from the National Hospital Ambulatory Care Survey between 1993 and 2005 and identified ED visits with diagnoses commonly caused by S. aureus, such as cellulitis, abscess and impetigo.

They found that infections of interest were diagnosed at 1.2 million visits in 1993, compared with 3.4 million visits in 2005. As a proportion of all ED visits, these infections were diagnosed 1.35% of the time in 1993 compared with 2.98% of the time in 2005.

When patients were prescribed antibiotics during these visits, antibiotics typically active against CA-MRSA were prescribed more frequently by 2005 (38%) than they had been from 1993 to 2001 (95% CI, 30-45; P<.001).

Our study shows that there is indeed an epidemic, Pallin said. We found that the rate of ED visits for skin and soft-tissue infections nearly tripled from 1993 to 2005. We also found that doctors are now treating skin and soft-tissue infections with antibiotics that are effective against CA-MRSA, when in the past, they didn’t.

Despite confirmation of an epidemic, the public should not be alarmed because the majority of the cases have been mild, according to Pallin. However, surveillance of staphylococci is important, and purulent skin infections should be cultured and treated as necessary.

Although we think trimethoprim-sulfamethoxazole is the best choice when antibiotics are needed for patients with abscesses, it’s important to understand that most abscesses should be treated with incision and drainage, not antibiotics, Pallin said. For skin infections that are not abscesses, we still don’t know if MRSA plays a role.

Mar

19

wineh.jpg
From Here :

Seeing these images of Amy Winehouse, and empathising with the way she might be feeling about her skin is easy as a Morgellons sufferer…
what is harder, much harder, no….impossible… is trying to imagine how she might be feeling with the media focusing on her every move, photographing her arms and face and publishing it for all to see.
I wish her luck and I wish her well.
However, I can not help but wonder, looking at these images if she might be suffering from Morgellons.
There are a few tell tale signs….and i’m looking beyond the “scratches”.

arm.jpg

casinos espana portalsistemas ruletaganar dinero real internetcasinos portales webruleta forojuego gratis pagina webjuegos para bajar gratisjugar keno gratisjuego instantaneo portal webjuego de casino,el juego de casino,descargar juego de casinoruleta portal webjuegos instantaneos internetjuego seguro portal webdorado hotel casinoganar dinero real paginas internetjuegos casino descargaganancia casino portales webjuego ruleta gratis para pcruleta suerte gratisjugar interactivo paginas internetjuego lineajuegos azar paginas internetonline gamingcasinos internacionales pagina internetjugar interactivo portal webcasino virtual portales internetbaccarat internettragaperras pagina webruleta americana onlinejugar casino onlinejuegos seguros webapuesta dinero paginas internetpremios portalesganancias casinos portales internetroulette onlinecasino dinero gratisjuego gratis lineajuegos instantaneos portal webmaquinas tragaperras portales internetcasino virtual paginas internetruleta pagina webtragaperras portales internetjuegue casino gratisplay kenodescargar ruleta gratistrucos casino onlinejuego instantaneo internetapostar internetjuegos azar paginas internet,juegos azar portales web,juegos azarjuego portal internet

Mar

14

What an arrogant and ill informed respose. I am shocked and upset.

The fact is, my GP is unable to clinically diagnose Lyme Borreliosis.

So at this stage, the informed patient, concerned about deteriorating health might make a request for a test.

My LB test came back from Southampton with a note …”question rising titers if recently bitten” .

My GP did not understand this sentence. I don’t understand this sentence.

Does anybody understand this sentence?

I have had more than 10 tick bites. So what are the “rising titers.” I was treated as being negative.

Typically, the majority of people bitten by an infected tick DO NOT develop a rash and there is no mention of the potential bacterial and viral co-infections which can be present, (with or without Lyme Borreliosis) in a tick’s saliva.

I must point out also that the vectors of Lyme Borreliosis are numerous, from the mouse to the deer.
How frustrating.

There are other inaccuracies and all are standard denials.

Has anybody even thought about this reply. It is beyond belief.

Prime Minister Gordon Brown, you should be ashamed.

From Here :

Details of Petition:

“That funding and resources be urgently made available and used to improve all aspects of the diagnosis, treatment, prevention, monitoring, public awareness and research of Lyme disease and its associated tick-borne infections, in order to improve the poor clinical outcome for people with these diseases in the UK.”

The Prime Minister’s Reply :

Clinicians in the UK have ready access to the best diagnostic tests available for Lyme disease. NHS diagnostic tests for Lyme disease conform to internationally agreed criteria and the tests are freely available within the NHS. Likewise, appropriate treatment with antibiotics is readily available for all those diagnosed with Lyme disease. Routine surveillance for Lyme disease is in place and was enhanced in 1996. Surveillance is undertaken by the Health Protection Agency (HPA). Its Lyme Borreliosis Specialist Diagnostic service provides not only diagnostic laboratory confirmation in support of clinical assessment but also provides advice and support to clinicians through its network of experts in infectious diseases, neurology, rheumatology and other specialities that have a particular interest in Lyme disease.

The internationally agreed criteria adopted by the UK for diagnostic tests for Lyme disease ensure that appropriate and standardised interpretation of tests is made across the UK. These criteria (Annex 1) are based upon stringent interpretation of serological tests for specific antibodies to Borrelia burgdorferi sensu lato, the causative agent of Lyme disease. Diagnostic testing is a two stage process: testing for the presence of antibodies to B. burgdorferi followed by specific immunoblot (Western blot) tests on all specimens that react in preliminary tests. The significance of the results is then carefully assessed in the light of the patient’s clinical signs and any history they have of exposure to ticks. It is accepted that diagnosis of Lyme disease is not straightforward and this is why it is particularly important for international authoritative experts, whose work in this field is peer-reviewed within the scientific community, to agree on validated testing methodologies.

The majority of people infected suffer only mild symptoms and will not require any treatment. Clinical symptoms are typically an initial raised red rash (erythema migrans) that spreads around the site of the tick bite. For those in whom illness develops, a short course of antibiotic treatment (using doxycycline or amoxicillin) is usually successful. On rare occasions, some people develop more serious symptoms. Treatment of late stage Lyme disease is also through antibiotics. However, long-term use of antibiotics is not indicated for the treatment of Lyme disease.

Sound surveillance for reporting of Lyme disease in people is in place in the UK. This surveillance system is based on laboratory confirmed reports and, as is stated above, was enhanced in 1996. However, it is accepted that there is under-reporting of Lyme disease, as a clear diagnosis is sometimes possible and appropriate antibiotic treatment given without recourse to laboratory confirmation, for example when clinical symptoms are obvious and the patient has recently been bitten by a tick. Such cases, as well as those where infection is asymptomatic or results in only mild symptoms that do not require medical attention, are not included within the national figures. Laboratory confirmed reports are received by the HPA and this enables an estimate of the rate of the disease to be calculated. As the majority of affected people suffer only mild symptoms and might miss or ignore the initial red rash and recover without recourse to their GP, making the disease notifiable would not detect these cases.

The number of cases of Lyme borreliosis diagnosed each year in the United Kingdom has increased steadily for a number of years, although the number of cases of neuroborreliosis diagnosed annually has remained steady since 2001. This increase is thought to be due to several factors, including increased awareness of the disease, greater access to diagnostic facilities, more sensitive diagnostic methods and the introduction of the enhanced surveillance scheme in 1996. Other factors include:

the increase in the number and geographical range of deer which are important hosts for feeding ticks;
greater public access to some parts of the countryside where Lyme borreliosis occurs, this has come about through changes in access legislation and increased interest in outdoor recreational activities; and
a succession of relatively mild winters which has allowed greater numbers of ticks to survive in the vegetation.
The HPA provides information about Lyme disease for both clinicians and the public on its website. The treatment guidelines for clinicians published by the Infectious Diseases Society of America in 2006 are authoritative and comprehensive, and similar to the recommendations of various European experts. They are available on the HPA website, together with published guidance for the diagnosis and treatment of neuroborreliosis, which is based on careful reviews of the scientific literature and is strongly recommended as valuable resources in the diagnosis and management of patients with suspected neuroborreliosis.

The HPA also works to increase awareness of Lyme disease through presentations to, and discussions with professional, recreational and special interest groups. For example, in spring 2007, before the start of the major tick feeding period, the HPA ran a national campaign to warn people of the risks associated with Lyme borreliosis and of ways to prevent infection. In addition, NHS Direct has published comprehensive information on the disease, including preventive measures, on its website.

The HPA has been at the forefront of close international collaboration, working with experts around the world on a range of issues to improve diagnostic tests and to promote evidence based treatments. International collaboration and research is fundamental to controlling and preventing disease at home. The UK can benefit from the research in countries where Lyme disease rates are much higher such as the USA and other countries in Europe.

The Department of Health does not consider that further research is needed at this time, as much is already known about diagnosis, treatment and mode of transmission of Lyme disease and other tick borne diseases. Lyme disease is already acknowledged as the most important vector borne disease in the UK, though the incidence rate is not high.

Mar

14

Oomycetes

March 14, 2008 | Leave a Comment

From here :

…..This shape ruled out candida which has septate hyphae and the pathogens Mucor and rhizopus since they contained chitin.

That pretty much left me with the oomycetes family. I was not happy to learn this. The Irish potato famine pathogen p.infestans, downy mildew, pythiosis, and saprolegnia were among the family along with 500 plus other varieties.

The oomycetes family is a freshwater mold by name but has only recently been understood to be a protist which mimics the shape of mold. They do not like salt.
In aquarium fish, there is a disease that is called Ich. In larger fish it is called saprolegnia and infests salmon and a few other varieties. These are more of the oomycetes family. All members of oomycetes have motile zoospores.

To shorten up this long story I will tell you that the fibers I have on my body were hollow, cellulose, fluorescent and were blue, red, and whitish as well as crystalline. This led me down the rabbit hole to bioengineered fibers containing pathogens.

I next focused on oomycetes pathogens that were being bioengineered into pesticides, and Lagenidium Giganteum came on the scene as a bioengineered Mosquito larvacide. It was touted to be harmless to mammals. It was in the form of “mycelium and oospores” to quote the pan pesticides data base. Sounds like fibers to me. A common practice in making biofibers is to add fluorescent marker dyes. Luciferase and Green fluorescent protein could account for the color and fluorescence Of these strands.

A short time later it was documented that a number of dogs had died from an emerging new oomycetes pathogen called Lagenidium Giganteum.

It presented in cutaneous lesions like pythiosis and then went systemic.
I called an expert in the field named Leonel Mendoza and asked him some questions about this emergent new pathogen.

He said it presented much like pythiosis but was even faster to become systemic. I asked him about human implications land he said “only dogs have it.” Since the disease, pythiosis he compared it to was zoonotic, I really believed that it could possibly be a human pathogen as well.
Just last week I was made aware of the article written by Amy Grooters that indeed the harmless mosquito pathogen Lagenidium Giganteum was now a human disease. There was never a mention of the mosquito pesticide in any of the illness reports. That same University had an ongoing research project on campus as well making bioengineered pesticides. Of course,once they found that pathogen, they knew what to look for. Colleges get grants for research from the government. No mention of their approved pesticide was ever mentioned. Go figure-
Arlene’e note: Isn’t this a dead givaway?

The Morgellons organization as well as a study by NUSPA both show the largest number of cases of this unknown fiber disease are reported from Texas, Florida, and California. These were the same three states that were doing the most extensive spraying with Laginex. Targets of choice included rice fields and soybean fields in addition to wetlands and theme parks. California even had a stronger strain of their own for use. That has now been discontinued without explanation.

There is an ELISA test that is available from Pan American Labs to detect antibodies from both pythium and lagenidium.

Physicians don’t even know it exists. I have not been able to get one of these tests. It is very frustrating to be in an HMO.

Cellulose and glucans (sugar) comprise the structure of oomycetes.
Humans have no enzymes with which to break down this pathogen in the human body.
The cellulose glucan resudue is a natural food for many insects. Infestation with insects can also add to the pathogen as well as bacteria.

I know that the form of lagenidium used for this spray is bioengineered and the cells of many other creatures have been used to make this product durable and give it longevity. In its natural state, lagenidium is a frail and scarce form of oomycetes. In its bioengineered state the product stays active for about a month as used, and will go into a encysted dormant survival phase which lasts up to 7 years. God only knows what the other types of cells were that are a part of this pathogen.

I am doing very well on the holistic medication I am taking. It has been of great help to deal with this disease as a fresh water protist.

The Mycoxan is superior for being able to penetrate the cell walls and kill the pathogen, research xanthones. The candex is a must because it contains the enzymes, cellulase, and hemicellulase which is in fact the enzyme from another variety of mold that is know to break down the cellulose residue. The copper and Guaco address and protist aspect of this disease.
I must say it has been great getting rid of all 30+ nonhealing lesions on my body and well as feeling much better. I will continue my regimen for a while though. It may take a while to break down all encysted spores and get them out of my body. From time to time I am still getting white hardened exudate coming out of my skin. It is a process but so far so good.

Mar

10

From Here :
I have had two knee replacements in the past three years. When the first knee was replaced, the incision did not heal properly and grew lesions on the scar that were filled with fibers. A year later the second knee was replaced and this time, Concord Hospital in Concord, New Hampshire made a series of pathology slides for Dr. Hildegarde Staninger who was at that time already researching Morgellon’s disease. She sent my pathology slides to Dr. Rahim Karjoo who made the following findings, which were later made public. The silicon and silicone photos are from my body. I have never had any implants or injections of silicone in my body. This silicon is a result of silicon-based nanotechnology.

Mar

5

BioWeopons

March 5, 2008 | Leave a Comment

From Here

Into a relatively innocuous bacterium responsible for a low-mortality pneumonia, Legionella pneumophila, Popov and his researchers spliced mammalian DNA that expressed fragments of myelin protein, the electrically insulating fatty layer that sheathes our neurons. In test animals, the pneumonia infection came and went, but the myelin fragments borne by the recombinant Legionella goaded the animals’ immune systems to read their own natural myelin as pathogenic and to attack it. Brain damage, paralysis, and nearly 100 percent mortality resulted: Popov had created a biological weapon that in effect triggered rapid multiple sclerosis. (Popov’s claims can be corroborated: in recent years, scientists researching treatments for MS have employed similar methods on test animals with similar results.)

Mar

1

From Here :

Fungal Sinusitis

What Is A Fungus? Fungi are plant-like organisms that lack chlorophyll. Since they do not have chlorophyll, fungi must absorb food from dead organic matter. Fungi share with bacteria the important ability to break down complex organic substances of almost every type (cellulose) and are essential to the recycling of carbon and other elements in the cycle of life. Fungi are supposed to “eat” only dead things, but sometimes they start eating when the organism is still alive. This is the cause of fungal infections; the treatment selected has to eradicate the fungus to be effective.

In the past 30 years, there has been a significant increase in the number of recorded fungal infections. This can be attributed to increased public awareness, new immunosuppressive therapies (medications such as cyclosporine that “fool” the body’s immune system to prevent organ rejection) and overuse of antibiotics (anti-infectives).

When the body’s immune system is suppressed, fungi find an opportunity to invade the body and a number of side effects occur. Because these organisms do not require light for food production, they can live in a damp and dark environment. The sinuses, consisting of moist, dark cavities, are a natural home to the invading fungi. When this occurs, fungal sinusitis results.

There Are Four Types Of Fungal Sinusitis:

Mycetoma Fungal Sinusitis produces clumps of spores, a “fungal ball,” within a sinus cavity, most frequently the maxillary sinuses. The patient usually maintains an effective immune system, but may have experienced trauma or injury to the affected sinus(es). Generally, the fungus does not cause a significant inflammatory response, but sinus discomfort occurs. The noninvasive nature of this disorder requires a treatment consisting of simple scraping of the infected sinus. An anti-fungal therapy is generally not prescribed.
Allergic Fungal Sinusitis (AFS) is now believed to be an allergic reaction to environmental fungi that is finely dispersed into the air. This condition usually occurs in patients with an immunocompetent host (possessing the ability to mount a normal immune response). Patients diagnosed with AFS have a history of allergic rhinitis, and the onset of AFS development is difficult to determine. Thick fungal debris and mucin (a secretion containing carbohydrate-rich glycoproteins) are developed in the sinus cavities and must be surgically removed so that the inciting allergen is no longer present. Recurrence is not uncommon once the disease is removed. Anti-inflammatory medical therapy and immunotherapy are typically prescribed to prevent AFS recurrence.

Note: A 1999 study published in the Mayo Clinic Proceedings asserts that allergic fungal sinusitis is present in a significant majority of patients diagnosed with chronic rhinosinusitis. The study found 96 percent of the study subjects with chronic rhinosinusitis to have a fungus in cultures of their nasal secretions. In sensitive individuals, the presence of fungus results in a disease process in which the body’s immune system sends eosinophils (white blood cells distinguished by their lobulated nuclei and the presence of large granules that attract the reddish-orange eosin stain) to attack fungi, and the eosinophils irritate the membranes in the nose. As long as fungi remain, so will the irritation.

Chronic Indolent Sinusitis is an invasive form of fungal sinusitis in patients without an identifiable immune deficiency. This form is generally found outside the US, most commonly in the Sudan and northern India. The disease progresses from months to years and presents symptoms that include chronic headache and progressive facial swelling that can cause visual impairment.

Microscopically, chronic indolent sinusitis is characterized by a granulomatous inflammatory infiltrate (nodular shaped inflammatory lesions). A decreased immune system can place patients at risk for this invasive disease.

Fulminant Sinusitis is usually seen in the immunocompromised patient (an individual whose immunologic mechanism is deficient either because of an immunodeficiency disorder or because it has been rendered so by immunosuppressive agents). The disease leads to progressive destruction of the sinuses and can invade the bony cavities containing the eyeball and brain.

The recommended therapies for both chronic indolent and fulminant sinusitis are aggressive surgical removal of the fungal material and intravenous anti-fungal therapy.

March 2008
M T W T F S S
« Feb   Apr »
 12
3456789
10111213141516
17181920212223
24252627282930
31  

Links